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Thiol-based antioxidants elicit mitochondrial oxidation via respiratory complex III

Excessive oxidation is widely accepted as a precursor to deleterious cellular function. On the other hand, an awareness of the role of reductive stress as a similar pathological insult is emerging. Here we report early dynamic changes in compartmentalized glutathione (GSH) redox potentials in living...

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Detalhes bibliográficos
Publicado no:Am J Physiol Cell Physiol
Main Authors: Kolossov, Vladimir L., Beaudoin, Jessica N., Ponnuraj, Nagendraprabhu, DiLiberto, Stephen J., Hanafin, William P., Kenis, Paul J. A., Gaskins, H. Rex
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2015
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4504939/
https://ncbi.nlm.nih.gov/pubmed/25994788
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00006.2015
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