טוען...
Exogenous and evoked oxytocin restores social behavior in the Cntnap2 mouse model of autism
Mouse models of neuropsychiatric diseases provide a platform for mechanistic understanding and development of new therapies. We previously demonstrated that knockout of the mouse homologue of CNTNAP2, in which mutant forms cause Cortical Dysplasia and Focal Epilepsy syndrome (CDFE), displays many fe...
שמור ב:
| הוצא לאור ב: | Sci Transl Med |
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| Main Authors: | , , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2015
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4498455/ https://ncbi.nlm.nih.gov/pubmed/25609168 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1126/scitranslmed.3010257 |
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