Autoimmunity to two forms of glutamate decarboxylase in insulin-dependent diabetes mellitus.
Insulin-dependent diabetes mellitus (IDDM) is thought to result from the autoimmune destruction of the insulin-producing beta cells of the pancreas. Years before IDDM symptoms appear, we can detect autoantibodies to one or both forms of glutamate decarboxylase (GAD65 and GAD67), synthesized from the...
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| 出版年: | J Clin Invest |
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| 主要な著者: | , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Clinical Investigation
1992
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC442846/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1370298/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI115573 |
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