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Porphyromonas gingivalis attenuates ATP-mediated inflammasome activation and HMGB1 release through expression of a nucleoside-diphosphate kinase

Many intracellular pathogens evade the innate immune response in order to survive and proliferate within infected cells. We show that Porphyromonas gingivalis, an intracellular opportunistic pathogen, uses a nucleoside-diphosphate kinase (NDK) homolog to inhibit innate immune responses due to stimul...

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Detalhes bibliográficos
Publicado no:Microbes Infect
Main Authors: Johnson, Larry, Atanasova, Kalina R., Bui, Phuong Q., Lee, Jungnam, Hung, Shu-Chen, Yilmaz, Özlem, Ojcius, David M.
Formato: Artigo
Idioma:Inglês
Publicado em: 2015
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4426005/
https://ncbi.nlm.nih.gov/pubmed/25828169
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.micinf.2015.03.010
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