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Increased surface expression of CD11b/CD18 (Mac-1) is not required for stimulated neutrophil adherence to cultured endothelium.

The mechanism whereby the human neutrophil membrane heterodimer, CD11b/CD18 (Mac-1, Mo1), mediates neutrophil adherence is not known. We studied the role of CD11b/CD18 surface expression in the promotion of neutrophil adhesiveness. We found that phorbol myristate acetate (PMA), calcium ionophore (A2...

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Detaylı Bibliyografya
Yayımlandı:J Clin Invest
Asıl Yazarlar: Vedder, N B, Harlan, J M
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Clinical Investigation 1988
Konular:
Online Erişim:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC442514/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/3278004/
https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI113372
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