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Variation in complement protein C1q is not a major contributor to cognitive impairment in Parkinson’s disease
Traditional dogma regarding the brain as an immune exempt organ has changed in recent years. New research has highlighted the role of the classical complement cascade in both synaptic elimination and function, driven largely by the role of the pathway initiating protein C1q. Given the links between...
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| Vydáno v: | Neurosci Lett |
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| Hlavní autoři: | , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Elsevier Scientific Publishers Ireland
2015
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4407898/ https://ncbi.nlm.nih.gov/pubmed/25817358 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neulet.2015.03.048 |
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