Rifampicin reduces advanced glycation end products and activates DAF-16 to increase lifespan in Caenorhabditis elegans

Advanced glycation end products (AGEs) are formed when glucose reacts nonenzymatically with proteins; these modifications are implicated in aging and pathogenesis of many age-related diseases including type II diabetes, atherosclerosis, and neurodegenerative disorders. Thus, pharmaceutical intervent...

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Опубликовано в: :Aging Cell
Главные авторы: Golegaonkar, Sandeep, Tabrez, Syed S, Pandit, Awadhesh, Sethurathinam, Shalini, Jagadeeshaprasad, Mashanipalya G, Bansode, Sneha, Sampathkumar, Srinivasa-Gopalan, Kulkarni, Mahesh J, Mukhopadhyay, Arnab
Формат: Artigo
Язык:Inglês
Опубликовано: BlackWell Publishing Ltd 2015
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Original Articles
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC4406675/
https://ncbi.nlm.nih.gov/pubmed/25720500
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acel.12327
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