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Actin cytoskeletal rearrangement and dysfunction due to activation of the receptor for advanced glycation end products is inhibited by thymosin beta 4
The receptor of advanced glycation end products (RAGE) is a cell-surface receptor that is a key factor in the pathogenesis of diabetic complications, including vascular disorders. Dysfunction of the actin cytoskeleton contributes to disruption of cell membrane repair in response to various type of e...
Gorde:
| Argitaratua izan da: | J Physiol |
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| Egile Nagusiak: | , |
| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
BlackWell Publishing Ltd
2015
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4405748/ https://ncbi.nlm.nih.gov/pubmed/25640761 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2014.287045 |
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