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Targeting CD36‐Mediated Inflammation Reduces Acute Brain Injury in Transient, but not Permanent, Ischemic Stroke
AIMS: The pathology of stroke consists of multiple pro‐death processes, and CD36 has been suggested as a multimodal target to reduce oxidative stress and inflammation in ischemic stroke. Using CD36‐deficient mice and SS‐31, a cell permeable tetrapeptide known to down‐regulate CD36 pathways, the curr...
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| Publicado no: | CNS Neurosci Ther |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
John Wiley and Sons Inc.
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4362808/ https://ncbi.nlm.nih.gov/pubmed/25216018 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cns.12326 |
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