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Early-onset metabolic syndrome in mice lacking the intestinal uric acid transporter SLC2A9
Excess circulating uric acid, a product of hepatic glycolysis and purine metabolism, often accompanies metabolic syndrome. However, whether hyperuricemia contributes to development of metabolic syndrome or is merely a by-product of other processes that cause this disorder has not been resolved. Addi...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | Nat Commun |
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| Κύριοι συγγραφείς: | , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
2014
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4348061/ https://ncbi.nlm.nih.gov/pubmed/25100214 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ncomms5642 |
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