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Antibacterial toxin colicin N and phage protein G3p compete with TolB for a binding site on TolA

Most colicins kill Escherichia coli cells by membrane pore formation or nuclease activity and, superficially, the mechanisms are similar: receptor binding, translocon recruitment, periplasmic receptor binding and membrane insertion. However, in detail, they employ a wide variety of molecular interac...

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Bibliografiset tiedot
Julkaisussa:Microbiology (Reading)
Päätekijät: Ridley, Helen, Lakey, Jeremy H.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Society for General Microbiology 2015
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4339652/
https://ncbi.nlm.nih.gov/pubmed/25536997
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1099/mic.0.000024
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