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P75 and phosphorylated c-Jun are differentially regulated in spinal motoneurons following axotomy in rats☆
The neurotrophin receptor (p75) activates the c-Jun N-terminal kinase (JNK) pathway. Activation of JNK and its substrate c-Jun can cause apoptosis. Here we evaluate the role of p75 in spinal motoneurons by comparing immunoreactivity for p75 and phosphorylated c-Jun (p-c-Jun), the production of JNK a...
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| Gepubliceerd in: | Neural Regen Res |
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| Hoofdauteurs: | , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Medknow Publications & Media Pvt Ltd
2012
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4296419/ https://ncbi.nlm.nih.gov/pubmed/25624831 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3969/j.issn.1673-5374.2012.26.001 |
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