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Reduction of Infarct Size by the Therapeutic Protein Tat-Ndi1 in vivo
Lethal myocardial ischemia-reperfusion injury has been attributed in part to mitochondrial respiratory dysfunction (including damage to Complex I) and the resultant excessive production of reactive oxygen species. Recent evidence has shown that Ndi1 (the single-subunit protein that in yeast serves t...
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| Yayımlandı: | J Cardiovasc Pharmacol Ther |
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| Asıl Yazarlar: | , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
2013
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4294554/ https://ncbi.nlm.nih.gov/pubmed/24367006 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/1074248413515750 |
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