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Calmodulin Mutations Associated with Long QT Syndrome Prevent Inactivation of Cardiac L-type Ca(2+) Currents and Promote Proarrhythmic Behavior in Ventricular Myocytes
Recent work has identified missense mutations in calmodulin (CaM) that are associated with severe early-onset long-QT syndrome (LQTS), leading to the proposition that altered CaM function may contribute to the molecular etiology of this subset of LQTS. To date, however, no experimental evidence has...
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| Publicado no: | J Mol Cell Cardiol |
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| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4262253/ https://ncbi.nlm.nih.gov/pubmed/24816216 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2014.04.022 |
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