Complement depletion accelerates the clearance of immune complexes from the circulation of primates.

Binding of immune complexes (IC) to erythrocytes in vitro is the result of interaction between C3b sites on the IC, and complement receptors type I (CRI) expressed on primate erythrocytes. Recent evidence indicates that primate erythrocytes can also rapidly bind large, preformed IC in vivo. This stu...

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Detaylı Bibliyografya
Yayımlandı:	J Clin Invest
Asıl Yazarlar:	Waxman, F J, Hebert, L A, Cornacoff, J B, VanAman, M E, Smead, W L, Kraut, E H, Birmingham, D J, Taguiam, J M
Materyal Türü:	Artigo
Dil:	Inglês
Baskı/Yayın Bilgisi:	American Society for Clinical Investigation 1984
Konular:	Research Article
Online Erişim:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC425300/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/6480828/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI111543
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Complement depletion accelerates the clearance of immune complexes from the circulation of primates.

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