Susceptibility to hepatocellular carcinoma is associated with genetic variation in the enzymatic detoxification of aflatoxin B1.

Aflatoxin B1 (AFB1) has been postulated to be a hepatocarcinogen in humans, possibly by causing p53 mutations at codon 249. AFB1 is metabolized via the phase I and II detoxification pathways; hence, genetic variation at those loci may predict susceptibility to the effects of AFB1. To test this hypot...

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Detalhes bibliográficos
Main Authors: McGlynn, K A, Rosvold, E A, Lustbader, E D, Hu, Y, Clapper, M L, Zhou, T, Wild, C P, Xia, X L, Baffoe-Bonnie, A, Ofori-Adjei, D
Formato: Artigo
Idioma:Inglês
Publicado em: 1995
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC42488/
https://ncbi.nlm.nih.gov/pubmed/7892276
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