S100A1 is released from ischemic cardiomyocytes and signals myocardial damage via Toll-like receptor 4

Members of the S100 protein family have been reported to function as endogenous danger signals (alarmins) playing an active role in tissue inflammation and repair when released from necrotic cells. Here, we investigated the role of S100A1, the S100 isoform with highest abundance in cardiomyocytes, w...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Rohde, David, Schön, Christoph, Boerries, Melanie, Didrihsone, Ieva, Ritterhoff, Julia, Kubatzky, Katharina F, Völkers, Mirko, Herzog, Nicole, Mähler, Mona, Tsoporis, James N, Parker, Thomas G, Linke, Björn, Giannitsis, Evangelos, Gao, Erhe, Peppel, Karsten, Katus, Hugo A, Most, Patrick
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Blackwell Publishing Ltd 2014
Aiheet:
Research Articles
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4203355/
https://ncbi.nlm.nih.gov/pubmed/24833748
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/emmm.201303498
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