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Thymosin-β(4) prevents cardiac rupture and improves cardiac function in mice with myocardial infarction
Thymosin-β(4) (Tβ4) promotes cell survival, angiogenesis, and tissue regeneration and reduces inflammation. Cardiac rupture after myocardial infarction (MI) is mainly the consequence of excessive regional inflammation, whereas cardiac dysfunction after MI results from a massive cardiomyocyte loss an...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Physiological Society
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4187393/ https://ncbi.nlm.nih.gov/pubmed/25015963 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00129.2014 |
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