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Acute hepatic ischemic-reperfusion injury induces a renal cortical “stress response,” renal “cytoresistance,” and an endotoxin hyperresponsive state

Hepatic ischemic-reperfusion injury (HIRI) is considered a risk factor for clinical acute kidney injury (AKI). However, HIRI's impact on renal tubular cell homeostasis and subsequent injury responses remain ill-defined. To explore this issue, 30–45 min of partial HIRI was induced in CD-1 mice....

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Detaylı Bibliyografya
Asıl Yazarlar: Zager, Richard A., Johnson, Ali C. M., Frostad, Kirsten B.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Physiological Society 2014
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4187044/
https://ncbi.nlm.nih.gov/pubmed/25080526
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00378.2014
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