Long-Term Ethanol and Corticosterone Co-Exposure Sensitize the Hippocampal CA1 Region Pyramidal Cells to Insult During Ethanol Withdrawal in an NMDA GluN2B Subunit-Dependent Manner

BACKGROUND: Chronic ethanol (EtOH) exposure produces neuroadaptations in NMDA receptor function and/or abundance and alterations in hypothalamic–pituitary–adrenal (HPA) axis functioning that contribute to neuronal excitation and neurotoxicity during ethanol withdrawal (EWD). Both EtOH and corticoste...

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Autors principals: Butler, Tracy R., Berry, Jennifer N., Sharrett-Field, Lynda J., Pauly, James R., Prendergast, Mark A.
Format: Artigo
Idioma:Inglês
Publicat: 2013
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4166417/
https://ncbi.nlm.nih.gov/pubmed/23889203
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acer.12195
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