Increased AP-1 and NF-κB activation and recruitment with the combination of the proinflammatory cytokines IL-1β, tumor necrosis factor alpha and IL-17 in rheumatoid synoviocytes

Ítems similars

• Combination of the proinflammatory cytokines IL-1, TNF-α and IL-17 leads to enhanced expression and additional recruitment of AP-1 family members, Egr-1 and NF-κB in osteoblast-like cells
  per: Granet, C, et al.
  Publicat: (2003)
• Effect of the combination of the proinflammatory cytokines IL-1, tumor necrosis factor-α and IL-17
  per: Miossec, P, et al.
  Publicat: (2003)
• Contribution of tumour necrosis factor α and interleukin (IL) 1ß to IL6 production, NF-κB nuclear translocation, and class I MHC expression in muscle cells: in vitro regulation with specific cytokine inhibitors
  per: Chevrel, G, et al.
  Publicat: (2005)
• IL-32, a proinflammatory cytokine in rheumatoid arthritis
  per: Joosten, Leo A. B., et al.
  Publicat: (2006)
• Addition of interleukin 1 (IL1) and IL17 soluble receptors to a tumour necrosis factor α soluble receptor more effectively reduces the production of IL6 and macrophage inhibitory protein-3α and increases that of collagen in an in vitro model of rheumatoid synoviocyte activation
  per: Chevrel, G, et al.
  Publicat: (2002)