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Simulations Suggest Pharmacological Methods for Rescuing Long-Term Potentiation
Congenital cognitive dysfunctions are frequently due to deficits in molecular pathways that underlie the induction or maintenance of synaptic plasticity. For example, Rubinstein-Taybi syndrome (RTS) is due to a mutation in cbp, encoding the histone acetyltransferase CREB-binding protein (CBP). CBP i...
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2014
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4162763/ https://ncbi.nlm.nih.gov/pubmed/25034337 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jtbi.2014.07.006 |
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