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A potassium leak channel silences hyperactive neurons and ameliorates status epilepticus
OBJECTIVE: To develop a constitutively active K(+) leak channel using TREK-1 (TWIK-related potassium channel 1; TREK-M) that is resistant to compensatory down-regulation by second messenger cascades, and to validate the ability of TREK-M to silence hyperactive neurons using cultured hippocampal neur...
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| 主要な著者: | , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2013
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4161023/ https://ncbi.nlm.nih.gov/pubmed/24299204 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/epi.12472 |
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