Yüklüyor......
Overexpression of Shox2 Leads to Congenital Dysplasia of the Temporomandibular Joint in Mice
Our previous study reported that inactivation of Shox2 led to dysplasia and ankylosis of the temporomandibular joint (TMJ), and that replacing Shox2 with human Shox partially rescued the phenotype with a prematurely worn out articular disc. However, the mechanisms of Shox2 activity in TMJ developmen...
Kaydedildi:
| Asıl Yazarlar: | , , , , , |
|---|---|
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
MDPI
2014
|
| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4159784/ https://ncbi.nlm.nih.gov/pubmed/25062348 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/ijms150813135 |
| Etiketler: |
Etiketle
Etiket eklenmemiş, İlk siz ekleyin!
|