Helicobacter pylori Induces AGS Cell Motility and Elongation via Independent Signaling Pathways

Helicobacter pylori induces motogenic and cytoskeletal responses in gastric epithelial cells. We demonstrate that these responses can be induced via independent signaling pathways that often occur in parallel. The cag pathogenicity island appears to be nonessential for induction of motility, whereas...

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Tallennettuna:
Bibliografiset tiedot
Päätekijät: Moese, Stefan, Selbach, Matthias, Kwok, Terry, Brinkmann, Volker, König, Wolfgang, Meyer, Thomas F., Backert, Steffen
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Microbiology 2004
Aiheet:
Molecular Pathogenesis
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC415712/
https://ncbi.nlm.nih.gov/pubmed/15155677
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/IAI.72.6.3646-3649.2004
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