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Overexpression of c-Met increases the tumor invasion of human prostate LNCaP cancer cells in vitro and in vivo
c-Met is a transmembrane tyrosine kinase receptor that may be activated by hepatocyte growth factor, an inducer of epithelial-mesenchymal transition (EMT), to regulate the associated downstream gene expression. This process is critical to cell migration in normal and pathological conditions. In the...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
D.A. Spandidos
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4156182/ https://ncbi.nlm.nih.gov/pubmed/25202379 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3892/ol.2014.2390 |
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