Redox Activation of DUSP4 by N-Acetyl Cysteine Protects Endothelial Cells from Cd(2+)-Induced Apoptosis

Redox imbalance is a primary cause for endothelial dysfunction (ED). Under oxidant stress, many critical proteins regulating endothelial function undergo oxidative modifications that lead to ED. Cellular levels of GSH, the primary reducing source in cells, can significantly regulate cell function vi...

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Hlavní autoři: Barajas-Espinosa, Alma, Basye, Ariel, Jesse, Erin, Yan, Haixu, Quan, David, Chen, Chun-An
Médium: Artigo
Jazyk:Inglês
Vydáno: 2014
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4146716/
https://ncbi.nlm.nih.gov/pubmed/24973647
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2014.06.016
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