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Interleukin-1β-Induced Wnt5a Enhances Human Corneal Endothelial Cell Migration through Regulation of Cdc42 and RhoA

Wnt5a can activate β-catenin-independent pathways for regulation of various cellular functions, such as migration, that play critical roles in wound repair. Investigation of Wnt5a signaling may help identify therapeutic targets for enhancing corneal endothelial wound healing that could provide an al...

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Detalles Bibliográficos
Main Authors: Lee, Jeong Goo, Heur, Martin
Formato: Artigo
Idioma:Inglês
Publicado: American Society for Microbiology 2014
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC4135619/
https://ncbi.nlm.nih.gov/pubmed/25022753
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.01572-13
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