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Zinc Potentiates GluK3 Glutamate Receptor Function by Stabilizing the Ligand Binding Domain Dimer Interface

Kainate receptors (KARs) play a key role in the regulation of synaptic networks. Here, we show that zinc, a cation released at a subset of glutamatergic synapses, potentiates glutamate currents mediated by homomeric and heteromeric KARs containing GluK3 at 10–100 μM concentrations, whereas it inhibi...

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Detalhes bibliográficos
Main Authors: Veran, Julien, Kumar, Janesh, Pinheiro, Paulo S., Athané, Axel, Mayer, Mark L., Perrais, David, Mulle, Christophe
Formato: Artigo
Idioma:Inglês
Publicado em: 2012
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4132841/
https://ncbi.nlm.nih.gov/pubmed/23141068
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2012.08.027
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