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Pharmacological modulation of heat shock factor 1 by antiinflammatory drugs results in protection against stress-induced cellular damage.

The activation of heat shock genes by diverse forms of environmental and physiological stress has been implicated in a number of human diseases, including ischemic damage, reperfusion injury, infection, neurodegeneration, and inflammation. The enhanced levels of heat shock proteins and molecular cha...

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Bibliografische Detailangaben
Veröffentlicht in:Proc Natl Acad Sci U S A
Hauptverfasser: Lee, B S, Chen, J, Angelidis, C, Jurivich, D A, Morimoto, R I
Format: Artigo
Sprache:Inglês
Veröffentlicht: National Academy of Sciences 1995
Schlagworte:
Online-Zugang:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC41308/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7638169/
https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.92.16.7207
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