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Resistance to cardiomyocyte hypertrophy in ae3( −/− ) mice, deficient in the AE3 Cl(−)/HCO(3)(−) exchanger
BACKGROUND: Cardiac hypertrophy is central to the etiology of heart failure. Understanding the molecular pathways promoting cardiac hypertrophy may identify new targets for therapeutic intervention. Sodium-proton exchanger (NHE1) activity and expression levels in the heart are elevated in many model...
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| Autori principali: | , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
BioMed Central
2014
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4120010/ https://ncbi.nlm.nih.gov/pubmed/25047106 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2261-14-89 |
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