Resistance to cardiomyocyte hypertrophy in ae3( −/− ) mice, deficient in the AE3 Cl(−)/HCO(3)(−) exchanger

BACKGROUND: Cardiac hypertrophy is central to the etiology of heart failure. Understanding the molecular pathways promoting cardiac hypertrophy may identify new targets for therapeutic intervention. Sodium-proton exchanger (NHE1) activity and expression levels in the heart are elevated in many model...

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Autores principales: Sowah, Daniel, Brown, Brittany F, Quon, Anita, Alvarez, Bernardo V, Casey, Joseph R
Formato: Artigo
Lenguaje:Inglês
Publicado: BioMed Central 2014
Materias:
Research Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC4120010/
https://ncbi.nlm.nih.gov/pubmed/25047106
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2261-14-89
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