Endothelial nitric oxide synthase is a regulator of hemodynamics in sepsis

Podobne knjige/članki

• Endothelial nitric oxide synthase deficiency and inducible nitric oxide synthase inhibition in the setting of septic cardiomyopathy
  od: Sandt, A Van de, et al.
  Izdano: (2009)
• Nitric oxide synthase 2-derived NO in endotoxemia is not sufficient to cause hemodynamic effects
  od: Brouckaert, P, et al.
  Izdano: (2008)
• Effects of inducible nitric oxide synthase and neuronal nitric oxide synthase inhibition on methicillin-resistant Staphylococcus aureus sepsis
  od: Bansal, Kamna, et al.
  Izdano: (2008)
• Endothelial Nitric Oxide Synthase and Superoxide Mediate Hemodynamic Initiation of Intracranial Aneurysms
  od: Liaw, Nicholas, et al.
  Izdano: (2014)
• Evaluation of the SOFA (Sepsis-related Organ Failure Assessment) Score in 303 consecutive patients of a medical intensive care unit
  od: Janssens, U, et al.
  Izdano: (1999)