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Mapping the architecture of the HIV-lTat circuit: A decision-making circuit that lacks bistability and exploits stochastic noise

Upon infection of a CD4(+) T cell, HIV-l appears to ‘choose’ between two alternate fates: active replication or a long-lived dormant statetermed proviral latency. A transcriptional positive-feedback loop generated by the HIV-l Tat protein appears sufficient to mediate this decision. Here, we describ...

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Hlavní autoři: Razooky, Brandon S., Weinberger, Leor S.
Médium: Artigo
Jazyk:Inglês
Vydáno: 2010
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4096296/
https://ncbi.nlm.nih.gov/pubmed/21167940
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ymeth.2010.12.006
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