Fibronectin fragments modulate monocyte VLA-5 expression and monocyte migration

To identify the mechanisms that cause monocyte localization in infarcted myocardium, we studied the impact of ischemia-reperfusion injury on the surface expression and function of the monocyte fibronectin (FN) receptor VLA-5 (α(5)β(1) integrin, CD49e/CD29). Myocardial infarction was associated with...

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Bibliografiset tiedot
Julkaisussa:J Clin Invest
Päätekijät: Trial, JoAnn, Baughn, Robert E., Wygant, James N., McIntyre, Bradley W., Birdsall, Holly H., Youker, Keith A., Evans, Alida, Entman, Mark L., Rossen, Roger D.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Clinical Investigation 1999
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Linkit:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC408518/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10449434/
https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI4824
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