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Insulin effects on glucose tolerance, hypermetabolic response, and circadian-metabolic protein expression in a rat burn and disuse model

Insulin controls hyperglycemia after severe burns, and its use opposes the hypermetabolic response. The underlying molecular mechanisms are poorly understood, and previous research in this area has been limited because of the inadequacy of animal models to mimic the physiological effects seen in hum...

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Detalhes bibliográficos
Main Authors: Pidcoke, Heather F., Baer, Lisa A., Wu, Xiaowu, Wolf, Steven E., Aden, James K., Wade, Charles E.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2014
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4080276/
https://ncbi.nlm.nih.gov/pubmed/24760998
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpregu.00312.2013
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