Transcytosis of cholera toxin subunits across model human intestinal epithelia.
Cholera toxin (CT) elicits a massive secretory response from intestinal epithelia by binding apical receptors (ganglioside GM1) and ultimately activating basolateral effectors (adenylate cyclase). The mechanism of signal transduction from apical to basolateral membrane, however, remains undefined. W...
Tallennettuna:
| Julkaisussa: | Proc Natl Acad Sci U S A |
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| Päätekijät: | , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
National Academy of Sciences
1995
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC40742/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7479732/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.92.22.10094 |
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