Suppression of retinal neovascularization in vivo by inhibition of vascular endothelial growth factor (VEGF) using soluble VEGF-receptor chimeric proteins.

The majority of severe visual loss in the United States results from complications associated with retinal neovascularization in patients with ischemic ocular diseases such as diabetic retinopathy, retinal vein occlusion, and retinopathy of prematurity. Intraocular expression of the angiogenic prote...

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Veröffentlicht in:	Proc Natl Acad Sci U S A
Hauptverfasser:	Aiello, L P, Pierce, E A, Foley, E D, Takagi, H, Chen, H, Riddle, L, Ferrara, N, King, G L, Smith, L E
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	National Academy of Sciences 1995
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC40630/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7479819/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.92.23.10457
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Suppression of retinal neovascularization in vivo by inhibition of vascular endothelial growth factor (VEGF) using soluble VEGF-receptor chimeric proteins.

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