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Biallelic expression of Tbx1 protects the embryo against developmental defects caused by increased Receptor Tyrosine Kinase signalling
BACKGROUND: 22q11.2 deletion syndrome (22q11DS) is the most common microdeletion syndrome in humans, characterised by cardiovascular defects such as interrupted aortic arch, outflow tract defects, thymus and parathyroid hypo- or aplasia and cleft palate. Heterozygosity of Tbx1, the mouse homologue o...
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| Main Authors: | , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4048740/ https://ncbi.nlm.nih.gov/pubmed/22674535 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/dvdy.23812 |
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