Traumatic Brain Injury In Vivo and In Vitro Contributes to Cerebral Vascular Dysfunction through Impaired Gap Junction Communication between Vascular Smooth Muscle Cells

Gap junctions (GJs) contribute to cerebral vasodilation, vasoconstriction, and, perhaps, to vascular compensatory mechanisms, such as autoregulation. To explore the effects of traumatic brain injury (TBI) on vascular GJ communication, we assessed GJ coupling in A7r5 vascular smooth muscle (VSM) cell...

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Bibliographische Detailangaben
Hauptverfasser: Yu, Guang-Xiang, Mueller, Martin, Hawkins, Bridget E., Mathew, Babu P., Parsley, Margaret A., Vergara, Leoncio A., Hellmich, Helen L., Prough, Donald S., DeWitt, Douglas S.
Format: Artigo
Sprache:Inglês
Veröffentlicht: Mary Ann Liebert, Inc. 2014
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Original Articles
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4047850/
https://ncbi.nlm.nih.gov/pubmed/24341563
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/neu.2013.3187
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