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Gain-of-function mutation in Gnao1: A murine model of epileptiform encephalopathy (EIEE17)?
G protein-coupled receptors strongly modulate neuronal excitability but there has been little evidence for G protein mechanisms in genetic epilepsies. Recently, four patients with epileptic encephalopathy (EIEE17) were found to have mutations in GNAO1, the most abundant G protein in brain, but the m...
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| Main Authors: | , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Springer US
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4042023/ https://ncbi.nlm.nih.gov/pubmed/24700286 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00335-014-9509-z |
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