First evidence of subclinical renal tubular injury during sickle-cell crisis

BACKGROUND: The pathophysiologic mechanisms classically involved in sickle-cell nephropathy include endothelial dysfunction and vascular occlusion. Arguments demonstrating that ischemia-reperfusion injury-related kidney damage might coincide with vaso-occlusive crisis (VOC) are lacking. METHODS: In...

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Main Authors: Audard, Vincent, Moutereau, Stéphane, Vandemelebrouck, Gaetana, Habibi, Anoosha, Khellaf, Mehdi, Grimbert, Philippe, Levy, Yves, Loric, Sylvain, Renaud, Bertrand, Lang, Philippe, Godeau, Bertrand, Galactéros, Frédéric, Bartolucci, Pablo
Formato: Artigo
Idioma:Inglês
Publicado: BioMed Central 2014
Assuntos:
Research
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC4006801/
https://ncbi.nlm.nih.gov/pubmed/24779676
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1750-1172-9-67
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