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Mechanism of copper transport at the blood–cerebrospinal fluid barrier: influence of iron deficiency in an in vitro model

Copper (Cu) is an essential trace element that requires tight homeostatic regulation to ensure appropriate supply while not causing cytotoxicity due to its strong redox potential. Our previous in vivo study has shown that iron deficiency (FeD) increases Cu levels in brain tissues, particularly in th...

Ausführliche Beschreibung

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Bibliographische Detailangaben
Hauptverfasser: Monnot, Andrew D, Zheng, Gang, Zheng, Wei
Format: Artigo
Sprache:Inglês
Veröffentlicht: 2012
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3982225/
https://ncbi.nlm.nih.gov/pubmed/22442359
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1258/ebm.2011.011170
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