The secretion of the Shigella flexneri Ipa invasins is activated by epithelial cells and controlled by IpaB and IpaD.

Eitemau Tebyg

• Nonpolar mutagenesis of the ipa genes defines IpaB, IpaC, and IpaD as effectors of Shigella flexneri entry into epithelial cells.
  gan: Ménard, R, et al.
  Cyhoeddwyd: (1993)
• Induction of Necrosis in Human Neutrophils by Shigella flexneri Requires Type III Secretion, IpaB and IpaC Invasins, and Actin Polymerization
  gan: François, Mathias, et al.
  Cyhoeddwyd: (2000)
• IpaD of Shigella flexneri Is Independently Required for Regulation of Ipa Protein Secretion and Efficient Insertion of IpaB and IpaC into Host Membranes
  gan: Picking, Wendy L., et al.
  Cyhoeddwyd: (2005)
• Deoxycholate Interacts with IpaD of Shigella flexneri in Inducing the Recruitment of IpaB to the Type III Secretion Apparatus Needle Tip
  gan: Stensrud, Kenneth F., et al.
  Cyhoeddwyd: (2008)
• Bile Salts Stimulate Recruitment of IpaB to the Shigella flexneri Surface, Where It Colocalizes with IpaD at the Tip of the Type III Secretion Needle
  gan: Olive, Andrew J., et al.
  Cyhoeddwyd: (2007)