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Ankyrin repeat and BTB/POZ domain containing protein-2 inhibits the aggregation of alpha-synuclein: Implications for Parkinson’s disease

Aggregation of α-synuclein is a pathological hallmark of sporadic or familial PD. However, the detailed molecular mechanism responsible for the aggregation of α-synuclein has not been properly explored. In the present study, we have identified a novel role of an anti-tumorigenic BTB/POZ domain conta...

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Detalles Bibliográficos
Main Authors: Roy, Avik, Pahan, Kalipada
Formato: Artigo
Idioma:Inglês
Publicado: 2013
Assuntos:
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC3929398/
https://ncbi.nlm.nih.gov/pubmed/24076025
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.febslet.2013.09.020
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