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Fatal eosinophilic myocarditis develops in the absence of IFNγ and IL17A
CD4(+) T cells play a central role in inflammatory heart disease, implicating a cytokine product associated with helper T cell effector function as a necessary mediator of this pathophysiology. IFNγ-deficient mice developed severe autoimmune myocarditis (EAM), in which mice are immunized with cardia...
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| 主要な著者: | , , , , , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2013
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3927983/ https://ncbi.nlm.nih.gov/pubmed/24048893 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1301282 |
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