Diesel exhaust particle-treated human bronchial epithelial cells upregulate Jagged-1 and OX40L in myeloid dendritic cells via TSLP()

Ambient particulate matter (PM), including diesel exhaust particles (DEP), promote the development of allergic disorders. Diesel exhaust particles increase oxidative stress and influence human bronchial epithelial cell (HBEC)-dendritic cell (DC) interactions via cytokines including thymic stromal ly...

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Hauptverfasser: Bleck, Bertram, Tse, Doris B., Gordon, Terry, Ahsan, Mohammad R., Reibman, Joan
Format: Artigo
Sprache:Inglês
Veröffentlicht: 2010
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Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3927452/
https://ncbi.nlm.nih.gov/pubmed/20974985
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1000719
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