Deficits in memory and hippocampal long-term potentiation in mice with reduced calbindin D28K expression.

The influx of calcium into the postsynaptic neuron is likely to be an important event in memory formation. Among the mechanisms that nerve cells may use to alter the time course or size of a spike of intracellular calcium are cytosolic calcium binding or "buffering" proteins. To consider t...

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Main Authors:	Molinari, S, Battini, R, Ferrari, S, Pozzi, L, Killcross, A S, Robbins, T W, Jouvenceau, A, Billard, J M, Dutar, P, Lamour, Y, Baker, W A, Cox, H, Emson, P C
Format:	Artigo
Sprog:	Inglês
Udgivet:	1996
Fag:	Research Article
Online adgang:	https://ncbi.nlm.nih.gov/pmc/articles/PMC38869/ https://ncbi.nlm.nih.gov/pubmed/8755597
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Deficits in memory and hippocampal long-term potentiation in mice with reduced calbindin D28K expression.

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