טוען...
MicroRNA-208a Increases Myocardial Fibrosis via Endoglin in Volume Overloading Heart
MicroRNA-208a (mir-208a) is essential for cardiac hypertrophy and fibrosis. Endoglin, a co-receptor of transforming growth factor-β is also essential for cardiac fibrosis. Endoglin has been shown to be a target of mir-208a in the in vitro mechanical stress model. Volume overload can lead to heart fa...
שמור ב:
| Main Authors: | , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Public Library of Science
2014
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3879305/ https://ncbi.nlm.nih.gov/pubmed/24392114 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0084188 |
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