The anticancer agent doxorubicin disrupts mitochondrial energy metabolism and redox balance in skeletal muscle

Lignende værker

• Mitochondrial glutathione depletion reveals a novel role for the pyruvate dehydrogenase complex as a key H(2)O(2) emitting source under conditions of nutrient overload
  af: Fisher-Wellman, Kelsey H., et al.
  Udgivet: (2013)
• Linking mitochondrial bioenergetics to insulin resistance via redox biology
  af: Fisher-Wellman, Kelsey H., et al.
  Udgivet: (2012)
• Pyruvate dehydrogenase complex and nicotinamide nucleotide transhydrogenase constitute an energy consuming redox circuit
  af: Fisher-Wellman, Kelsey H., et al.
  Udgivet: (2015)
• Flux through mitochondrial redox circuits linked to nicotinamide nucleotide transhydrogenase generates counterbalance changes in energy expenditure
  af: Smith, Cody D., et al.
  Udgivet: (2020)
• Targeted overexpression of mitochondrial catalase protects against cancer chemotherapy-induced skeletal muscle dysfunction
  af: Gilliam, Laura A. A., et al.
  Udgivet: (2016)